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However, different mechanisms have been proposed depending on the type of viral infections, ranging from an imbalance in redox homeostasis with increased ROS production, oxidative stress, and decreased antioxidant enzymes [47] to decreased glutathione peroxidase and glutathione reductase levels [48]

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In controlled laboratory settings it is studied for its interaction with angiogenic signalling, principally VEGFR2 pathway activity, and for its modulation of nitric-oxide synthesis