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The subjects receiving most of their energy as fat had a lesser IGF-I response to GH, whereas subjects receiving the same caloric intake as carbohydrate demonstrated greater increases in IGF-I to GH stimulation (70, 71)

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Moreover, Glu transporter expression in the cerebral cortex of AD patients and VGLUT1 significantly decreased, possibly resulting in poor clearance of glutamic acid in the synaptic cleft, leading to excitatory toxicity